[1] |
Ma S, Evans RG, Iguchi N, et al. Sepsis-induced acute kidney injury:a disease of the microcirculation[J].Microcirculation,2019,26(2):e12483.
|
[2] |
殷志颖,马四清.慢性低氧环境下微血管增生的生理机制及病理生理学特征[J/CD].中华临床医师杂志(电子版),2022,16(4):372-375.
|
[3] |
马四清.高原慢性低氧暴露下的急性呼吸窘迫综合征:更应关注右心功能[J].解放军医学杂志,2022,47(4):321-325.
|
[4] |
Poston JT, Koyner JL. Sepsis associated acute kidney injury[J].BMJ,2019(364):k4891.
|
[5] |
何宗钊,邓莉,马四清,等.不同海拔汉族健康人大循环及微循环特征的对比[J].中国应用生理学杂志,2021,37(4):371-376.
|
[6] |
赵思涵,李红艳.低氧诱导因子1α 在炎症中的作用研究进展[J].中国免疫学志,2020,36(22):2809-2814.
|
[7] |
Rajaee A, Barnett R, Cheadle WG. Pathogen - and danger -associated molecular patterns and the cytokine response in sepsis[J].Surg Infect(Larchmt),2018,19(2):107-116.
|
[8] |
Singer M, Deutschman CS, Seymour CW, et al. The third international consensus definitions for sepsis and septic shock(sepsis-3)[J].JAMA,2016,315(8):801-810.
|
[9] |
Emlet DR, Shaw AD, Kellum JA. Sepsis - associated AKI:epithelial cell dysfunction[J].Semin Nephrol,2015,35(1):85-95.
|
[10] |
Bhargava P, Schnellmann RG. Mitochondrial energetics in the kidney[J].Nat Rev Nephrol,2017,13(10):629-646.
|
[11] |
Gómez H,Jin K, Kellum JA. The role of energy regulation in the tubular epithelial cell response to sepsis[J]. Nephron, 2015, 131(4):255-258.
|
[12] |
Gómez H, Kellum JA, Ronco C. Metabolic reprogramming and tolerance during sepsis-induced AKI[J]. Nat Rev Nephrol, 2017,13(3):143-151.
|
[13] |
La Rovere RM, Roest G, Bultynck G,et al.Intracellular Ca(2+)signaling and Ca(2+) microdomains in the control of cell survival,apoptosis and autophagy[J].Cell Calcium,2016,60(2):74-87.
|
[14] |
李静,赵双平.脓毒症相关性肾损伤[J].医学综述,2019,25(2):307-311,316.
|
[15] |
Bulluck H, Hausenloy DJ. Modulating NAD metabolism to prevent acute kidney injury[J].Nat Med,2018,24(9):1306-1307.
|
[16] |
Van Aelst LNL, Arrigo M, Placido R et al. Acutely decom -pensated heart failure with preserved and reduced ejection fraction present with comparable haemodynamic conges-tion[J].Eur J Heart Fail,2018,20(4):738-747.
|
[17] |
Méndez AB, Azancot MA, Olivella A, et al. New aspects in cardiorenal syndrome and HFpEF[J].Clin Kidney J,2022,15(10):1807-1815.
|
[18] |
Rangaswami J, Bhalla V, Blair JEA,et al. Cardiorenal syn-drome:classification,pathophysiology,diagnosis,and treat ment strategies:a scientific statement from the American Heart Association[J].Circulation,2019,139(16):e840-e878.
|
[19] |
Giam B, Kaye DM, Rajapakse NW. Role of renal oxidative stress in the pathogenesis of the cardiorenal syndrome[J]. Heart Lung Circ,2016,25(8):874-880.
|
[20] |
Monard C, Meersch-Dini M, Joannidis M. When the kidneys hurt,the other organs suffer[J]. Intensive Care Med, 2023, 49(2): 233-236.
|
[21] |
林霖,周荣斌.关于脓毒症休克早期目标导向治疗的再评价及研究进展[J].中国临床医生杂志,2016,44(2):29-31.
|
[22] |
马黎霞.脓毒血症液体管理现状与研究进展[J].中华实用诊断与治疗杂志,2016,30(3):222-224.
|
[23] |
张建起,石蕊,张芯,等.高原环境对心血管系统的影响及其防治措施[J].武警医学,2022,33(9):812-817.
|
[24] |
万有栋,朱瑞雪,高艳霞,等.休克患者液体复苏:如何避免液体过负荷[J/CD]. 中西医结合心血管病电子杂志, 2016, 4(19):6-7.
|
[25] |
姜志钊,刘玉琪,任建安.液体治疗降阶梯策略在腹腔脓毒性休克中的应用进展[J]. 中华危重病急救医学, 2020, 32(11):1403-1408.
|
[26] |
Zampieri FG, Bagshaw SM, Semler MW. Fluid therapy for critically ill adults with sepsis: a review[J]. JAMA, 2023, 329(22):1967-1980.
|
[27] |
Si-Qing MA, Shao-Hua P, Zong-Zhao HE, et al.Changes of microcirculation in healthy volunteers and patients with septic shock in Xining[J].中国应用生理学杂志,2016,32(6):533-539.
|
[28] |
金灵燕,范开亮,方华,等.脓毒症休克血管活性药物研究进展[J].现代临床医学,2017,43(4):243-245,250.
|
[29] |
Bai X,Yu W,Ji W,et al. Early versus delayed administration of norepinephrine in patients with septic shock[J]. Crit Care, 2014,18(5):532.
|
[30] |
丁显飞,万有栋,孙谋,等.脓毒性休克中血管活性药物的应用进展[J].中西医结合心血管病电子杂志,2016,4(19):13-15.
|
[31] |
李秋红.小剂量血管加压素联合去甲肾上腺素对感染性休克患者血流动力学及肾功能的影响[J].现代医学与健康研究(电子版),2021,5(1):69-71.
|
[32] |
林文利.连续性肾脏替代治疗对脓毒血症合并急性肾损伤患者脏器功能及存活率影响研究[J].陕西医学杂志,2019,48(1):22-24.
|
[33] |
吴相伟, 叶继辉, 孙敏, 等. CRRT 启动时机与脓毒症相关性AKI 患者预后的关系[J]. 中华危重病急救医学, 2020, 32(11):1352-1355.
|
[34] |
金仁华,沈骁,孙加奎,等.脓毒症急性肾损伤的肾脏替代治疗[J].内科急危重症杂志,2022,28(5):404-408.
|